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Pulseless electrical activity (PEA) and asystole | NCLEX-RN | Khan Academy

-[Voiceover] Ventricular
asystole and pulseless electrical activity are two
types of cardiac arrest, meaning the heart has stopped. In both of these, a patient
doesn’t have a pulse, meaning that they’re not pumping blood to the rest of the body, and
that’s why both of these conditions are absolutely fatal unless they’re corrected immediately. So in ventricular asystole, there’s no electrical
activity in the heart. That there’s no electrical
activity, that means that the ventricle walls
aren’t contracting. And that’s what asystole basically means. A means no, and systole implies ventricular contractions. So no ventricular contractions. Again, no electrical activity means no ventricular contractions, means no cardiac output,
or in other words, there’s no blood flowing
to the rest of the body. So there’s no cardiac output. And without cardiac output, you’re not going to have a pulse. So anybody who has ventricular asystole will not have a pulse. On an EKG, this looks like a flat line because there’s no electrical activity to cause any movement on an EKG. And this is the flat
line that you hear about on movies and TV shoes, and they say, “The patient’s flatlining!” It’s asystole. And then there’s pulseless
electrical activity. And this is known as PEA. In PEA, there is
electrical activity on EKG. However, it doesn’t result in a pulse. And the electrical
activity you see on an EKG could be something that
normally produces a pulse, such as normal sinus
rhythm, or even heart block, or sinus bradycardia, but for some reason there’s not a pulse. Now how could this be? Well, in a normal heart, the heart’s electrical activity causes the muscle cells to contract. So you have action
potentials that propagate or went through the heart, and they’ll lead to muscle contraction. And this relationship between electrical activity and mechanical contraction is called electro-mechanical coupling. However, when the heart is under extreme stress, so say the heart’s
been deprived of oxygen for a long time, the
system gets disconnected. So, even though there’s
electrical activity, it’s not going to lead to contractions. Because we’ve disconnected
the system, and this is called electro-mechanical uncoupling. So despite the fact that cells can undergo and propagate action potentials, the action potentials don’t result in muscle contraction. Another reason why you
could have electrical activity without a pulse is because there could be something blocking the heart. Now the heart has a sac outlining it called the pericardium. And in some cases, this
might be full of blood. And if it is full of
blood, it’s going to press down on the heart, and
the heart is not going to have any room to pump. This is called tamponade,
or cardiac tamponade. And again, this is the
condition where the heart is constricted by this fluid
filled sac around the heart, and the heart can’t pump. So even though you can
have action potentials and electrical activity in the heart, you’re not going to be able to pump, and you won’t have a pulse. Okay, so I’m sure you’ve seen in movies or TV shows,
when someone flat lines or has asystole, and
someone else comes running into the room, puts paddles on the chest, and yells, “Clear!” In this case, they’re
defibrillating the patient, meaning they’re providing electrical shock to the heart, hopefully
to convert the heart back to a normal rhythm. However, this is a common misconception. We never shock asystole
or PEA for that matter. Defibrillation only works on very specific abnormal cardiac rhythms that can potentially be reversed
with an electric shock, and these are called shockable rhythms. Shockable rhythms include
ventricular fibrillation, where the walls of the
ventricles are spasming, and therefore they can’t contract, and they’re not going to circulate blood to the rest of the body, and pulseless ventricular tachycardia, meaning that there’s some sort of abnormal conduction in the ventricles that cause the ventricles to pump at a dangerously high rate. So neither of these has a pulse. And just a side note, we never shock anyone with a pulse. Something else to note is that even though ventricular fibrillation
and pulseless v tach meet the criteria for PEA,
and that there’s electrical activity but no pulse, we don’t typically classify them as PEA. These two arrhythmias
are a class of their own because we treat them differently. They’re shockable rhythms. PEA is considered electric
activity without a pulse. That’s not v fib or pulseless v tach. Now how do we treat asystole and PEA? Asystole and PEA are
considered nonshockable rhythms, meaning providing a shock won’t likely restore a normal rhythm. How do we treat nonshockable rhythms? We start with cardiopulmonary
resuscitation, or CPR, where we alternate chest
compressions with some sort of artificial
breathing for the patient, whether that be mouth-to-mouth, or doing oxygen through some sort of mask. We’ll also administer
vasoconstrictive medications. Now, vasoconstrictive means
that we’re constricting the vasos, or the vessels. So these drugs constrict
the blood vessels. And equally important
with any patient who is pulseless, we have to consider any factors that could be reversed that might be contributing to the cardiac arrest. And you can remember which factors to consider by the mnemonic Hs and Ts, which we’re going to go
over in just one second. So what are the Hs and Ts? Remember, they’re potentially
reversible conditions that could be causing or even contributing to the cardiac arrest. So, if any patient in
cardiac arrest has any of these factors, we want
to try to reverse it, or fix it, to help save them. Now, something to note
is that a lot of these Hs and Ts are conditions
where there’s some sort of lack of adequate blood
circulating through the body, or adequate oxygen delivery to the body, including the heart. Heart cells need oxygen
to function properly. So if there’s not enough
oxygen getting to the heart, then the heart cells aren’t happy, and they quit working properly. The first H we’re going to talk about is hypovolemia, so hypo means low, and volemia refers to volume. So this basically means low blood volume. Usually, from excessive bleeding. In hypovolemia, not enough
blood is circulating, so not enough oxygen is getting around. The next H is hypoxia. That means that there’s inadequate oxygen supplied to the body. And hypoxia can be due to many things, such as drowning or even a heart attack. So there’s inadequate
oxygen getting to the body, to the heart, and to the brain. The next H is hydrogen ions. And hydrogen ions
basically means acidosis, meaning that the body’s PH is too low. And acidosis often results from long periods of hypoxia. We also need to consider
a person’s potassium level, because hypo and hyperkalemia can lead to cardiac arrest. Now, hypokalemia means that there’s too low potassium, and hyperkalemia means the potassium level is too high. Potassium plays a really
important role in maintaining normal electrical conduction in the heart. So you can imagine that
if the levels are too high or too low, this will disrupt the heart’s electrical conduction system. In anyone with cardiac
arrest that comes in, we’re going to check their glucose level, because hypoglycemia,
or low blood glucose, can lead to cardiac arrest, and this is something that’s easily fixed. And the last H we’re
going to talk about is hypothermia, or low body temperature. And typically, we think
of hypothermia as a temperature less than
95 degrees Fahrenheit or 35 degrees Celsius. So as a person’s core temperature drops, the heart’s pacemaker
cells fire less and less, and eventually, the heart can stop. Okay, so those are the Hs. Now, let’s move on to the Ts. We need to consider toxins, and toxins include both
prescription medications and street drugs. If someone comes in
because of cardiac arrest due to a toxin, there
might be a reversible agent that will help reverse
the effects of the toxins, and can help save the patient. The second T is tamponade, and that’s something we just talked about. And we’re referring to cardiac tamponade. Like we said, this is
a condition where blood fills the space that lines the heart, or the pericardial sac, and
this constricts the heart and makes it a lot harder
for the heart to pump, and sometimes the heart
can’t even pump at all. And if the heart’s not
pumping, no blood circulating. And one way I like to think about it is to think about doing a jumping jack. Now, imagine doing that
jumping jack underwater. It’s a lot harder to do
a jumping jack underwater because all this pressure’s around your arms and legs. Likewise, it’s harder for
the heart to pump with all the added pressure surrounding it. And the next T is something called tension pneumothorax. So in the chest wall, the lungs are surrounded by a plural lining. So there’s a space created
called the plural space between the lungs and the chest wall. In a tension pneumothorax, air can somehow enter this plural space. And this is usually because of some sort of trauma to the chest. And what happens is, air
enters this plural space, but it can’t leave, and
as more and more air enters the space, it crushes the lung, and even pushes the lung
and the heart to the side. A crushed or collapsed
lung is not going to be able to move oxygen very easily, and the heart can stop. And the last T we’re going to talk about is thrombosis,
basically meaning blood clot. In the case of cardiac arrest, we’re concerned about a blood clot to the coronary artery, or an artery that supplies the heart with oxygenated blood, or we’re concerned about
a clot in the lungs, and that’s known as a pulmonary embolism. So a pulmonary embolism
is a clot in the lung. So clots in either the heart or the lungs can lead to severe oxygen depletion, and eventually lead to cardiac arrest.

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